Vol 9: A Defective TLR4 Signaling for IFN- Expression Is Responsible for the Innately Lower Ability of BALB-c Macrophages to Produce NO in Response to LPS as Compared to C57BL-6.Report as inadecuate



 Vol 9: A Defective TLR4 Signaling for IFN- Expression Is Responsible for the Innately Lower Ability of BALB-c Macrophages to Produce NO in Response to LPS as Compared to C57BL-6.


Vol 9: A Defective TLR4 Signaling for IFN- Expression Is Responsible for the Innately Lower Ability of BALB-c Macrophages to Produce NO in Response to LPS as Compared to C57BL-6. - Download this document for free, or read online. Document in PDF available to download.

Download or read this book online for free in PDF: Vol 9: A Defective TLR4 Signaling for IFN- Expression Is Responsible for the Innately Lower Ability of BALB-c Macrophages to Produce NO in Response to LPS as Compared to C57BL-6.
This article is from PLoS ONE, volume 9.AbstractC57BL-6 mice macrophages innately produce higher levels of NO than BALB-c cells when stimulated with LPS.
Here, we investigated the molecular events that account for this intrinsic differential production of NO.
We found that the lower production of NO in BALB-c is not due to a subtraction of L-argini

Author: Oliveira, Luciana S.; de Queiroz, Nina M.
G.
P.; Veloso, Laura V.
S.; Moreira, Thais G.; Oliveira, Fernanda S.; Carneiro, Matheus B.
H.; Faria, Ana M.


Source: https://archive.org/





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